Traumatic iritis: Difference between revisions

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==Management==
==Management==
#Cycloplegics (eg. Homatropine 5%, or cyclopentolate 2%, 3 times per day until ophthalmology follow-up): paralyzes the ciliary body resulting in a nonreactive, dilated pupil
#PO analgesia
#PO analgesia
#Steroids in consult with optho (rule out infection first)
#Cycloplegics paralyze the ciliary body resulting in a nonreactive and dilated pupil, preventing synechiae, progression of flare, ciliary spasm pain<ref>Alexander KL, Dul MW, Lalle PA, Magnus DE. Onofrey B. Optometric Clinical Practice Guideline: Care of the Patient with Anterior Uveitis. St. Louis, MO: American Optometric Association; 1994:3-29.</ref>
##Homatropine 5% BID-TID
##Cyclopentolate 2% TID
##Scopolamine 0.25% BID
#Steroids in consult with optho
##Rule out infection first and avoid corneal epithelial defect
##Prednisolone acetate 0.5-1% QID
#If secondary glaucoma as complication, may use timolol 0.5% BID if no contraindication


==Disposition==
==Disposition==

Revision as of 21:39, 24 July 2016

Background

  • Classically blunt trauma: contusion and spasm of ciliary body and iris
  • May occur in any traumatic injury[1]
  • Iridocyclitis because both the iris and the underlying ciliary body are inflamed
  • Iritis
    • HLA-B27 antigenic marker also strongly linked to spondyloarthritis such as ankylosing spondylitis

Clinical Features

  • Often delayed presentation after traumatic event, typically 24-48 hrs
  • Eye pain, especially if not relieved by topical anesthetic[2]
  • Decreased visual acuity in affected eye
  • Photophobia (direct and consensual)
  • Sluggish pupil
  • Hypopyon (severe cases): leukocytic exudate in anterior chamber
  • Cell & flare (anterior chamber reaction)
    • “Cell:” individual cells floating in the anterior chamber (look like dust specks)
    • “Flare:" protein floating in the anterior chamber from inflamed blood vessels. (smoke)
    • May deposit onto corneal endothelium as keratic precipitates[3]
  • Vossius' ring on anterior lens capsule
    • Due to concussive force of posterior iris onto lens
    • Depositing pigment in the pattern of a miosed pupil onto anterior lens
  • IOP may be increased due to inflammation, damage to ciliary body, or circumferential synechial formation[4]

Differential Diagnosis

Unilateral red eye

^Emergent diagnoses ^^Critical diagnoses

Acute Vision Loss (Noninflamed)

Emergent Diagnosis

Diagnosis

  • Clinical diagnosis

Management

  1. PO analgesia
  2. Cycloplegics paralyze the ciliary body resulting in a nonreactive and dilated pupil, preventing synechiae, progression of flare, ciliary spasm pain[5]
    1. Homatropine 5% BID-TID
    2. Cyclopentolate 2% TID
    3. Scopolamine 0.25% BID
  3. Steroids in consult with optho
    1. Rule out infection first and avoid corneal epithelial defect
    2. Prednisolone acetate 0.5-1% QID
  4. If secondary glaucoma as complication, may use timolol 0.5% BID if no contraindication

Disposition

Follow up with optho in 24-48 hours

See Also

References

  1. Augsburger JJ, Corrêa ZM. Chapter 19. Ophthalmic Trauma. In: Riordan-Eva P, Cunningham, Jr. ET, eds. Vaughan &amp;amp;amp;amp;amp; Asbury's General Ophthalmology. 18th ed. New York, NY: McGraw-Hill; 2011:371-382.
  2. Reidy JJ. Section 08: External Disease and Cornea. Basic and Clinical Science Course. San Francisco, CA: American Academy of Ophthalmology; 2012: 363.
  3. Bartley GB, Liesegang TJ. Essentials of Ophthalmology. Philadelphia, PA: JB Lippincott Company; 1992:156-157.
  4. Trevor-Roper PD, Curran PV. The Eye and Its Disorders. Boston, MA: Blackwell Scientific Publications; 1984:489-507.
  5. Alexander KL, Dul MW, Lalle PA, Magnus DE. Onofrey B. Optometric Clinical Practice Guideline: Care of the Patient with Anterior Uveitis. St. Louis, MO: American Optometric Association; 1994:3-29.