Pulmonary edema: Difference between revisions

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*Non-cardiogenic
*Non-cardiogenic
**Hypertensive crisis.  
**Hypertensive crisis.  
*[[Negative pressure pulmonary edema]] (Upper airway obstruction)
*[[Negative pressure pulmonary edema]]  
**Upper airway obstruction
**Reexpansion edema
*Neurogenic causes
*Neurogenic causes
**[[Seizures]]
**[[Seizures]]
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**[[Pulmonary contusion]]
**[[Pulmonary contusion]]
**Aspiration
**Aspiration
**Reexpansion edema (post large volume thoracocentesis, resolution of pneumothorax, post decortication, removal of endobronchial obstruction, effectively a form of negative pressure pulmonary oedema.
*Immersion pulmonary edema
*Reperfusion injury, i.e. postpulmonary thromboendartectomy or lung transplantation
*Immersion pulmonary edema[7][8]
*Iatrogenic fluid overload
*Iatrogenic fluid overload
**Multiple blood transfusions
**Multiple blood transfusions
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*Hantavirus pulmonary syndrome
*Hantavirus pulmonary syndrome
*[[High altitude pulmonary edema]]
*[[High altitude pulmonary edema]]
Envenomation, such as with the venom of Atrax robustus[11]


==Diagnosis==
==Diagnosis==

Revision as of 21:00, 24 March 2015

Background

Causes

Diagnosis

  • Crackles
  • Respiratory distress

Differential Diagnosis

Acute dyspnea

Emergent

Non-Emergent

Treatment

  1. CPAP/BiPAP with PEEP 6-8; titrate up to PEEP of 10-12
  2. Nitroglycerin
    1. Dosing Options
      1. Sublingual 0.4 mg q5min
      2. Nitropaste (better bioavailability than oral Nitroglycerin)
      3. Intravenous: 0.1mcg/kg/min - 5mcg/kg/min

Generally start IV Nitroglycerin 50mcg/min and titrate rapidly (150mcg/min or higher)to symptom relief

  • If NTG fails to reduce BP consider nitroprusside (reduces both preload and afterload) or ACE-inhibitiors (preload reducer)
  • After pt improves titrate down NTG as enaliprilat (0.625 - 1.25mg IV) or captopril are started
  • Morphine is no longer recommended do to increased morbidity

See Also

References