Perioral dermatitis: Difference between revisions

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==Background==
==Background==
{{Skin anatomy background images}}
*90% female (increasing in males)
*90% female (increasing in males)


===Etiology===
===Etiology===
*Topical or inhaled steroids
*[[topical steroids|Topical]] or [[inhaled corticosteroid]]s
*use of cosmetics,
*use of cosmetics
*physical (UVB, heat, wind),
*physical (UVB, heat, wind)
*microbiological (fusiform spirilla, candida)
*microbiological (fusiform spirilla, [[candida]])
*hormonal factors (premenstrual deterioration, use of oral contraceptives)  
*hormonal factors (premenstrual deterioration, use of oral contraceptives)  
*GI tract abnormalities (malabsorption)
*GI tract abnormalities (malabsorption)
*emotional stress.
*Emotional stress
 
{{Dermatitis types}}


==Clinical Features==
==Clinical Features==
[[File:ADC 2008 335.jpg|thumb]]
[[File:ADC 2008 335.jpg|thumb]]
*papular, vesiculopapular and papulopustular lesions on erythematous base (Acneiform)
*papular, vesiculopapular and papulopustular [[rash|lesions]] on erythematous base (Acneiform)
**confluent aspect, follicular
**confluent aspect, follicular
*location perioral, limited to skin
*location perioral, limited to skin
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==Differential Diagnosis==
==Differential Diagnosis==
*Rosacea
*Rosacea
*Seborrheic Dermatitis
*[[Seborrheic dermatitis]]
*Acne vulgaris
*Acne vulgaris
*Facial demodicosis
*Facial demodicosis
*Lupus milliaris
*[[Lupus]] milliaris
*Polymorphous light reaction
*Polymorphous light reaction
*Contact dermatitis
*[[Contact dermatitis]]
*Haber syndrome
*Haber syndrome
*Granulomatous periorificial dermatitis
*Granulomatous periorificial dermatitis
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*Discontinue suspected topicals
*Discontinue suspected topicals
*Rebound reaction
*Rebound reaction
**hydrocortisone cuts down reaction violence
**[[hydrocortisone]] cuts down reaction violence
*Corticosteroid dependence - psychological follow up
*[[Corticosteroid]] dependence - psychological follow up
*Suppression of follicular bacterial infection
*Suppression of follicular bacterial infection
**Lipophilic tetracyclines 100-250mg/d for 3-4 months
**Lipophilic [[tetracyclines]] 100-250mg/d for 3-4 months
**no response - Isotretinoin
**no response - Isotretinoin
**Children - oral, topical Erythromycin, Metronidazole
**Children - oral, topical [[erythromycin]], [[metronidazole]]
*Antipruritics containing no corticosteroids
*Antipruritics containing no corticosteroids



Latest revision as of 17:28, 11 December 2024

Background

Normal dermal anatomy.
  • 90% female (increasing in males)

Etiology

  • Topical or inhaled corticosteroids
  • use of cosmetics
  • physical (UVB, heat, wind)
  • microbiological (fusiform spirilla, candida)
  • hormonal factors (premenstrual deterioration, use of oral contraceptives)
  • GI tract abnormalities (malabsorption)
  • Emotional stress

Dermatitis Types

Clinical Features

ADC 2008 335.jpg
  • papular, vesiculopapular and papulopustular lesions on erythematous base (Acneiform)
    • confluent aspect, follicular
  • location perioral, limited to skin
    • typically respects rim around and 3-5 mm under lower lip, circular

Differential Diagnosis

Oral rashes and lesions

Evaluation

  • Usually clinical

Management

  • Discontinue suspected topicals
  • Rebound reaction
  • Corticosteroid dependence - psychological follow up
  • Suppression of follicular bacterial infection
  • Antipruritics containing no corticosteroids

Complications

  • Disfiguring scars - emotional
  • Rebound effect, chronic course
  • Lupus-like PD - dermal infiltrate, scarring
    • Yellowish discoloration after diascopy

See Also

References

  • Ljubojević et al. "Perioral dermatitis" Acta Dermatovenerol Croat. 2008;16(2):96-100