ST segment elevation: Difference between revisions

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**80% of inferior STEMI have ST depression in anterior leads
**80% of inferior STEMI have ST depression in anterior leads
***However, may represent reciprocal changes OR posterior STEMI
***However, may represent reciprocal changes OR posterior STEMI
==See Also==
*[[ECG (Main)]]
*[[ACS - ECG]]


==Source==
==Source==

Revision as of 03:14, 13 January 2014

Background

  • Its presence must be explained (there is no "nonspecific ST elevation")
  • Not a specific marker for STEMI

DDX

  1. Myocardial Infarct (STEMI)
  2. Wellens' Syndrome
  3. Coronary spasm (eg, Prinzmetal's angina)
  4. Drugs (eg, cocaine, crack, meth)
  5. Pericarditis
  6. LV aneurysm
  7. Early repolarization
  8. LBBB
  9. LVH
  10. Misc
    1. Meds: TCA, digoxin
    2. RV pacing (appears as LBBB)
    3. Hyperkalemia
    4. Hypothermia
      1. "Osborn J waves"
    5. Brugada Syndrome

Early Repolarization versus STEMI

  • Early Repolarization suggested by:
    • ST elevation <4mm
    • Notched J point
    • Concave or saddle-back ST morphology
    • No reciprocal changes

LVH versus STEMI

  • Difficult to exclude MI in pt with LVH (pt already at risk for MI)
  • Best aid is prior ECG or serial ECGs

LBBB versus STEMI

See Sgarbossa's Criteria

STEMI

  • ST elevation in those leads that reflect the distribution of a single coronary artery
    • RCA: II, III, aVF (inferior)
    • LAD: V1-V4 (antero-septal)
    • Circumflex: V5-V6, I, aVL (lateral)
    • Dominant left circulation: infero-lateral
  • ST morphology
    • Concave up versus convex
      • Convex has Sp 97%, Sn 77%
  • Look for reciprocal ST depression
    • 35% of anterior STEMI have ST depression in inferior leads
    • 80% of inferior STEMI have ST depression in anterior leads
      • However, may represent reciprocal changes OR posterior STEMI

See Also

Source

  • ST Elevation Lecture, Dr. Niemann, Harbor-UCLA