Calcific tendinitis
Background
- Self-limiting disorder of calcium hydroxyapatite deposition within one or more tendons of the rotator cuff
- Usually spontaneous, not necessarily caused by trauma or overuse
- With time, the calcium undergoes painful resorption with subsequent tendon healing
- Middle-aged patients 30-50 years are most commonly affected (rarely seen in patients >70yrs), and women are moreso affected[1]
- Adhesive capsulitis is most common complication
- Inflammatory reaction may exhibit warmth, swelling, or erythema that may mimic septic arthritis
Clinical Features
Precalcific phase
- Fibrocartilaginous metaplasia of the tendon, leading to chondrocyte formation
- Pain-free
Calcification phase
- Formative phase
- Characterized by cell-mediated calcific deposits
- +/- Shoulder pain
- Resting phase
- Lacks inflammation or vascular infiltration
- +/- pain
- Resorptive phase (1-2 wks)
- Characterized by phagocytic resorption and vascular infiltration
- Most painful phase; due to inflammation in the resorptive process, increase in intratendinous pressure, or subacromial impingement
- Pain may be similar to rotator cuff tendinopathy; usually at rest, worse at night and with laying on affected side
- Leakage of calcium crystals onto the bursa may lead to sudden dramatic pain
- Any shoulder motion reproduces significant pain; shoulder impingement tests are usually positive
- TTP over proximal humerus near tendinous insertion of rotator cuff; may also be near AC joint or deltoid
Post-calcific phase
- Calcific deposits have been resorbed; tendon returns to normal
- Variable levels of pain and shoulder dysfunction
Differential Diagnosis
Shoulder and Upper Arm Diagnoses
Traumatic/Acute:
- Shoulder Dislocation
- Clavicle fracture
- Humerus fracture
- Scapula fracture
- Acromioclavicular joint injury
- Glenohumeral instability
- Rotator cuff tear
- Biceps tendon rupture
- Triceps tendon rupture
- Septic joint
Nontraumatic/Chronic:
- Rotator cuff tear
- Impingement syndrome
- Calcific tendinitis
- Adhesive capsulitis
- Biceps tendinitis
- Subacromial bursitis
- Cervical radiculopathy
Refered pain & non-orthopedic causes:
- Referred pain from
- Neck
- Diaphragm (e.g. gallbladder disease)
- Brachial plexus injury
- Axillary artery thrombosis
- Thoracic outlet syndrome
- Subclavian steal syndrome
- Pancoast tumor
- Myocardial infarction
- Pneumonia
- Pulmonary embolism
Evaluation
- Imaging
- Plain films will show calcification in the tendon(s) of the rotator cuff
- Ultrasound is sensitive for detecting calcifications
- May show hyperechoic areas and posterior acoustic shadowing
- Can simultaneously detect rotator cuff tears or bursitis
Management
- Nonoperative management is successful in most cases
- NSAIDs, oral and topical
- Physical therapy for rotator/glenohumeral strength and ROM
- Stretch and strengthening
- Avoid immobilization
- Rest shoulder in abduction on back of a chair as soon as tolerable
- Sleep with pillow beneath axilla
- Avoid immobilization
- Subacromial corticosteroid injection
Disposition
- Primary care referral within 1wk
- If chronic, may consider ortho referral for extracorporeal shock wave therapy, ultrasound-guided lavage, or operative management
See Also
References
- ↑ Merolla G, Singh S, Paladini P, Porcellini G. Calcific tendinitis of the rotator cuff: state of the art in diagnosis and treatment. J Orthop Traumatol. 2016 Mar;17(1):7-14. doi: 10.1007/s10195-015-0367-6. Epub 2015 Jul 12. PMID: 26163832; PMCID: PMC4805635.
- ↑ Kim MS, Kim IW, Lee S, Shin SJ. Diagnosis and treatment of calcific tendinitis of the shoulder. Clin Shoulder Elb. 2020 Nov 27;23(4):210-216. doi: 10.5397/cise.2020.00318. PMID: 33330261; PMCID: PMC7726362.
- ↑ Catapano, Michael, Robinson, David M., Schowalter, Sean and McInnis, Kelly C.. "Clinical evaluation and management of calcific tendinopathy: an evidence-based review" Journal of Osteopathic Medicine, vol. 122, no. 3, 2022, pp. 141-151. https://doi.org/10.1515/jom-2021-0213