Beer potomania syndrome: Difference between revisions
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==Background== | ==Background== | ||
The pathophysiology involves the inability to excrete sufficient free water, based on a loss of normal renal urea gradients. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline | The pathophysiology involves the inability to excrete sufficient free water, based on a loss of normal renal urea gradients. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline | ||
==Diagnosis== | ==Diagnosis== | ||
#a history of chronic alcohol ingestion (in a hypotonic form) | |||
#protein malnutrition | |||
#signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia | |||
#no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc. | |||
==Work-Up== | |||
Chem 10 | |||
==DDx== | |||
Insert | |||
Insert | |||
==Treatment== | |||
Insert | |||
==Disposition== | |||
Insert | Insert | ||
==See Also== | ==See Also== | ||
Tox: ETOH Intoxication | Tox: ETOH Intoxication | ||
Tox: ETOH Withdrawl | Tox: ETOH Withdrawl | ||
==Source== | ==Source== | ||
Va Med. 1989 Jun;116(6):270-1. Beer potomania syndrome in an alcoholic. Harrow AS. | Va Med. 1989 Jun;116(6):270-1. Beer potomania syndrome in an alcoholic. Harrow AS. | ||
[[Category:FEN]] | [[Category:FEN]] | ||
Revision as of 19:59, 13 March 2011
Background
The pathophysiology involves the inability to excrete sufficient free water, based on a loss of normal renal urea gradients. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline
Diagnosis
- a history of chronic alcohol ingestion (in a hypotonic form)
- protein malnutrition
- signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia
- no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc.
Work-Up
Chem 10
DDx
Insert
Treatment
Insert
Disposition
Insert
See Also
Tox: ETOH Intoxication
Tox: ETOH Withdrawl
Source
Va Med. 1989 Jun;116(6):270-1. Beer potomania syndrome in an alcoholic. Harrow AS.