Traumatic iritis: Difference between revisions

(dispo)
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==Background==
==Background==
*Blunt trauma -> contusion and spasm of ciliary body and iris
*Blunt trauma: contusion and spasm of ciliary body and iris
*Iridocyclitis because both the iris and the underlying ciliary body are inflamed
*Iritis
**HLA-B27 antigenic marker also strongly linked to spondyloarthritis such as ankylosing spondylitis


==Clinical Features==
==Clinical Features==
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*Sluggish pupil
*Sluggish pupil
*Cell & flare
*Cell & flare
*Hypopyon (severe cases)
**“Cell:” individual cells floating in the anterior chamber (look like dust specks)
**“Flare:" protein floating in the anterior chamber from inflamed blood vessels. (smoke)
*Hypopyon (severe cases): leukocytic exudate in anterior chamber


==Treatment==
==Treatment==
#Cycloplegics (eg. homotyptine 5%)
#Cycloplegics (eg. homotyptine 5%): paralyzes the ciliary body resulting in a nonreactive, dilated pupil
#PO analgesia
#PO analgesia
#Steroids in consult with optho
#Steroids in consult with optho (rule out infection first)


==Dispo==
==Dispo==

Revision as of 03:14, 13 January 2015

Background

  • Blunt trauma: contusion and spasm of ciliary body and iris
  • Iridocyclitis because both the iris and the underlying ciliary body are inflamed
  • Iritis
    • HLA-B27 antigenic marker also strongly linked to spondyloarthritis such as ankylosing spondylitis

Clinical Features

  • Eye pain
  • Decreased visual acuity in affected eye
  • Photophobia (direct and consensual)
  • Sluggish pupil
  • Cell & flare
    • “Cell:” individual cells floating in the anterior chamber (look like dust specks)
    • “Flare:" protein floating in the anterior chamber from inflamed blood vessels. (smoke)
  • Hypopyon (severe cases): leukocytic exudate in anterior chamber

Treatment

  1. Cycloplegics (eg. homotyptine 5%): paralyzes the ciliary body resulting in a nonreactive, dilated pupil
  2. PO analgesia
  3. Steroids in consult with optho (rule out infection first)

Dispo

F/u with optho in 24-48 hours

Source

  • PEER VIII Q&A