Respiratory alkalosis: Difference between revisions
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*[[Aspirin (Salicylate) Toxicity]] | *[[Aspirin (Salicylate) Toxicity]] | ||
*Progesterone/[[pregnancy]] | *Progesterone/[[pregnancy]] | ||
*Liver disease | *[[hepatic failure|Liver disease]] | ||
*[[CVA]] or other central cause | *[[CVA]] or other central cause | ||
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==References== | ==References== | ||
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[[Category:FEN]] | [[Category:FEN]] | ||
[[Category:Pulmonary]] | [[Category:Pulmonary]] | ||
[[Category:Toxicology]] | [[Category:Toxicology]] | ||
Latest revision as of 16:43, 29 September 2019
Background
Clinical Features
- Hyperventilation
Differential Diagnosis
- Asthma
- PE
- DKA
- Anxiety
- Hypoxia
- early Sepsis/fever/Pneumonia
- Hyperthyroid
- Sympathomimetics
- Aspirin (Salicylate) Toxicity
- Progesterone/pregnancy
- Liver disease
- CVA or other central cause
Evaluation
- alkalemia = pH >7.42
- respiratory alkalosis = pCO2 <38
- May lead to Hypocalcemia, Hypokalemia
- Check for a concurrent acid/base disturbance
- always check for an AG
- for every 10mm pCO2 <40, HCO3 expected to decrease by 1-3.5 mEq
- if HCO3 < 24 - (40-pCO2)/10 x 2.5 (+/-1) then there is a superimposed primary metabolic acidosis
- if HCO3 > 24 - (40-pCO2)/10 x 2.5 (+/-1) then there is a primary metabolic alkalosis, or acute respiratory alkalosis without time for metabolic compensation