ECG Basics: Difference between revisions

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==General==
== General ==
#1 small box = 1mm = 0.04 sec = 40 mili-seconds
#5 small boxes = 1 big box = 5mm = 0.2 sec = 200 mili-seconds


==Intervals==
*1 small box = 1mm = 0.04 sec = 40 miliseconds
ECG Seconds mm (sm boxes)
*5 small boxes = 1 big box = 5mm = 0.2 sec = 200 miliseconds


P 0.10 2.5
== Intervals ==


PR 0.12-0.20 3-5
{| border="1" cellspacing="1" cellpadding="1" style="width: 500px; "
|-
| Interval
| Time (s)
| boxes
|-
| PR
| 0.12 - 0.20
| 3-5
|-
| QRS
| .06 - 0.10
| 1.5-2.5
|-
| QTc
| <0.44
| N/A
|}


QRS 0.06-0.10 1.5-2.5
<span class="Apple-style-span" style="font-size: 18px; font-weight: bold; ">Axis</span>


Q <0.04 1
*QT 0.33-0.42 8.25-10.5
QTc <0.44 NA
==Axis==
#axis and ventricular hypertrophy cannot be measured correctly in presence of BBBs....
#axis and ventricular hypertrophy cannot be measured correctly in presence of BBBs....
#Anterior hemiblock-left axis deviationgreater than-45 to -60. and small q in 1 and avl. RS in 2,3, avf. Intrinsicoid deflection in avl greater than .045.
#Anterior hemiblock-left axis deviationgreater than-45 to -60. and small q in 1 and avl. RS in 2,3, avf. Intrinsicoid deflection in avl greater than .045.
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#Horizontal axis-find isoeletric chest lead. If shifted to V1 or V2 then counterclockwise rotation. If shifted to V5 or V6 then clockwise rotation.
#Horizontal axis-find isoeletric chest lead. If shifted to V1 or V2 then counterclockwise rotation. If shifted to V5 or V6 then clockwise rotation.


==Q waves==
== Q waves ==
 
#sig Q waves are @ least 1 mm (1 box) wide or @ least 1/3 of the entire QRS amplitude.
#sig Q waves are @ least 1 mm (1 box) wide or @ least 1/3 of the entire QRS amplitude.
#Early Repolarization:
#Early Repolarization:
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##limb leads may also have ste, rarely >2 mm
##limb leads may also have ste, rarely >2 mm


==T waves==
== T waves ==
 
#T waves are normally upright in 1,2,V3 to V6. Negative in AVR.
#T waves are normally upright in 1,2,V3 to V6. Negative in AVR.
##if T wave is greater than 2/3 height of R wave it is abnormal.
##if T wave is greater than 2/3 height of R wave it is abnormal.


==RAE==
== RAE ==
 
#P amplitude >2.5 mm in II.
#P amplitude >2.5 mm in II.
#Rarely isolated finding (usu rvh/rad too).
#Rarely isolated finding (usu rvh/rad too).
#large diphasic p wave in V1. Right atrial hyper.-initial component is larger in V1 than V6 :tall p wave, bigger than 2.5 boxes in limb leads.
#large diphasic p wave in V1. Right atrial hyper.-initial component is larger in V1 than V6&nbsp;:tall p wave, bigger than 2.5 boxes in limb leads.
#p-mitrale-m notched p wave in leads 1 and 2. Greater than .12 seconds.
#p-mitrale-m notched p wave in leads 1 and 2. Greater than .12 seconds.


==LAE==
== LAE ==
 
#Biphasic P in V1 w/ wide, deep terminal component, >1mm depth & wide.
#Biphasic P in V1 w/ wide, deep terminal component, >1mm depth & wide.
#Left atrial hyper.-terminal component is larger than .04 sec.
#Left atrial hyper.-terminal component is larger than .04 sec.


==LVH==
== LVH ==
 
#Sum of S in V1 or V2 & R in V5 or V6 is >35mm.
#Sum of S in V1 or V2 & R in V5 or V6 is >35mm.
#Sum of highest R & deepest S in precord is >45mm
#Sum of highest R & deepest S in precord is >45mm
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#R in AVL of >12mm
#R in AVL of >12mm
#L precordial leads may show ST depression & twi= LV strain pattern
#L precordial leads may show ST depression & twi= LV strain pattern
#LVH-s wave in V1 or V2 (biggest) plus R in V5 or V6 (biggest) is greater than 35mm. LAD-with slightly wide QRS. Or r in avl greater than 11mm, r in 1 is greater than 12mm, or R in AVf is greater than 20mm.
#LVH-s wave in V1 or V2 (biggest) plus R in V5 or V6 (biggest) is greater than 35mm. LAD-with slightly wide QRS. Or r in avl greater than 11mm, r in 1 is greater than 12mm, or R in AVf is greater than 20mm.
 
== RVH ==


==RVH==
#Dominant R in V1 >7mm (also seen in WPW, RBBB, post MI, & nml var.)
#Dominant R in V1 >7mm (also seen in WPW, RBBB, post MI, & nml var.)
#RSR in V1 w/ QRS < 0.12
#RSR in V1 w/ QRS < 0.12
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#Less sens. & spec. than LVH, usu nl ecg
#Less sens. & spec. than LVH, usu nl ecg
#RVH-R wave greater than S in V1, but gets progressively smaller from V1 to V6. S wave persists in V5 and V6. RAD with slightly wide QRS.
#RVH-R wave greater than S in V1, but gets progressively smaller from V1 to V6. S wave persists in V5 and V6. RAD with slightly wide QRS.
#(note: R :S ratio greater than 1 also in: 1.RBBB 2.WPW type A. 3. Post. Wall MI. 4.kids.)
#(note: R&nbsp;:S ratio greater than 1 also in: 1.RBBB 2.WPW type A. 3. Post. Wall MI. 4.kids.)
 
== RBBB ==


==RBBB==
#QRS > 0.12 in limb leads (all qrs intervals should be measured in limb leads)
#QRS > 0.12 in limb leads (all qrs intervals should be measured in limb leads)
#triphasic QRS (RSR') in ant precord leads (V1-V3), often w/ st dep & twi in these leads
#triphasic QRS (RSR') in ant precord leads (V1-V3), often w/ st dep & twi in these leads
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#V1 must have a positive complex.
#V1 must have a positive complex.


==LBB==
== LBBB ==
 
#QRS > 0.12 in limb leads, w/ lg, broad , notched or slurred R waves in lat precord leads (V5-V6) & lead I & avL, the st seg is usu depressed & twi in these leads!
#QRS > 0.12 in limb leads, w/ lg, broad , notched or slurred R waves in lat precord leads (V5-V6) & lead I & avL, the st seg is usu depressed & twi in these leads!
#can cause lad, ste seen in ant leads but can call ant mi in rbb!
#can cause lad, ste seen in ant leads but can call ant mi in rbb!
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#broad monomorphic S waves in V1, may have a small r wave.
#broad monomorphic S waves in V1, may have a small r wave.


==LASH==
== LASH ==
 
(caused by CAD, valv. dis., cong. dis., cardiomyop., myocard.)
(caused by CAD, valv. dis., cong. dis., cardiomyop., myocard.)


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#exclude other cause of L axis (habitus, Inf. MI, hyperK, Vent. pre-excitation)
#exclude other cause of L axis (habitus, Inf. MI, hyperK, Vent. pre-excitation)


==L Post Inf Hemiblock==
== L Post Inf Hemiblock ==
 
(USU. organic heart dis.)
(USU. organic heart dis.)


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#(-w/ combined blocks cons. pacing only if sxs w/ bradyarrhythmias)
#(-w/ combined blocks cons. pacing only if sxs w/ bradyarrhythmias)


==Source ==
== Source ==
 
9/09 DONALDSON (adapted from Niemann, Lampe, Pani)
9/09 DONALDSON (adapted from Niemann, Lampe, Pani)


[[Category:Cards]]
<br/>[[Category:Cards]] <br/><br/>

Revision as of 00:55, 8 April 2011

General

  • 1 small box = 1mm = 0.04 sec = 40 miliseconds
  • 5 small boxes = 1 big box = 5mm = 0.2 sec = 200 miliseconds

Intervals

Interval Time (s) boxes
PR 0.12 - 0.20 3-5
QRS .06 - 0.10 1.5-2.5
QTc <0.44 N/A

Axis

  1. axis and ventricular hypertrophy cannot be measured correctly in presence of BBBs....
  2. Anterior hemiblock-left axis deviationgreater than-45 to -60. and small q in 1 and avl. RS in 2,3, avf. Intrinsicoid deflection in avl greater than .045.
  3. posterior hemiblock-right axis deviation and S1Q3.
  4. Axis-check lead 1 and AVF..If both are positive then axis is normal .
  5. Find isoelectric lead. Axis is perpendicular.
  6. Horizontal axis-find isoeletric chest lead. If shifted to V1 or V2 then counterclockwise rotation. If shifted to V5 or V6 then clockwise rotation.

Q waves

  1. sig Q waves are @ least 1 mm (1 box) wide or @ least 1/3 of the entire QRS amplitude.
  2. Early Repolarization:
    1. STE most prominent in lat precord leads (V4-6) but no reciprocal chngs. T waves here usu broad, tall (usu > 5mm) & upright.
    2. limb leads may also have ste, rarely >2 mm

T waves

  1. T waves are normally upright in 1,2,V3 to V6. Negative in AVR.
    1. if T wave is greater than 2/3 height of R wave it is abnormal.

RAE

  1. P amplitude >2.5 mm in II.
  2. Rarely isolated finding (usu rvh/rad too).
  3. large diphasic p wave in V1. Right atrial hyper.-initial component is larger in V1 than V6 :tall p wave, bigger than 2.5 boxes in limb leads.
  4. p-mitrale-m notched p wave in leads 1 and 2. Greater than .12 seconds.

LAE

  1. Biphasic P in V1 w/ wide, deep terminal component, >1mm depth & wide.
  2. Left atrial hyper.-terminal component is larger than .04 sec.

LVH

  1. Sum of S in V1 or V2 & R in V5 or V6 is >35mm.
  2. Sum of highest R & deepest S in precord is >45mm
  3. R wave in V6 > 18mm
  4. R in AVL of >12mm
  5. L precordial leads may show ST depression & twi= LV strain pattern
  6. LVH-s wave in V1 or V2 (biggest) plus R in V5 or V6 (biggest) is greater than 35mm. LAD-with slightly wide QRS. Or r in avl greater than 11mm, r in 1 is greater than 12mm, or R in AVf is greater than 20mm.

RVH

  1. Dominant R in V1 >7mm (also seen in WPW, RBBB, post MI, & nml var.)
  2. RSR in V1 w/ QRS < 0.12
  3. This dx usu also w/ RAE or strain (ST dep w/ twi in V1-V3).
  4. Less sens. & spec. than LVH, usu nl ecg
  5. RVH-R wave greater than S in V1, but gets progressively smaller from V1 to V6. S wave persists in V5 and V6. RAD with slightly wide QRS.
  6. (note: R :S ratio greater than 1 also in: 1.RBBB 2.WPW type A. 3. Post. Wall MI. 4.kids.)

RBBB

  1. QRS > 0.12 in limb leads (all qrs intervals should be measured in limb leads)
  2. triphasic QRS (RSR') in ant precord leads (V1-V3), often w/ st dep & twi in these leads
  3. assoc w/ org heart dz (cant cause rad on own)
  1. QRS greater than .12.
  2. slurred s in 1 and V6.
  3. RSR' in V1 with R' taller than R.
  4. V1 must have a positive complex.

LBBB

  1. QRS > 0.12 in limb leads, w/ lg, broad , notched or slurred R waves in lat precord leads (V5-V6) & lead I & avL, the st seg is usu depressed & twi in these leads!
  2. can cause lad, ste seen in ant leads but can call ant mi in rbb!
  1. duration equals .12sec
  2. broad monomorphic R waves in 1 and V6 without Q waves.
  3. broad monomorphic S waves in V1, may have a small r wave.

LASH

(caused by CAD, valv. dis., cong. dis., cardiomyop., myocard.)

  1. Axis < -45 (L axis) w/QRS <0.10s
  2. deep S in II, III, and AVF
  3. exclude other cause of L axis (habitus, Inf. MI, hyperK, Vent. pre-excitation)

L Post Inf Hemiblock

(USU. organic heart dis.)

  1. R axis (>110) w/ QRS < 0.10s
  2. R waves in II, III, AVF.
  3. Exclude other causes (COPD, RVH, Lat MI)
  4. (-w/ combined blocks cons. pacing only if sxs w/ bradyarrhythmias)

Source

9/09 DONALDSON (adapted from Niemann, Lampe, Pani)